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Sustained effects of rapidly acting antidepressants require BDNF-dependent MeCP2 phosphorylation

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Our findings, in turn, suggest that these effects are similarly manifested in patients with CUD

The peptide achieves its effects through multiple interconnected signaling pathways rather than binding to a single receptor

Prevention of HGF nitration-dysfunction offers novel potential to suppress the progression of age-related disorders of muscle homeostasis including muscle atrophy and impaired regeneration accompanied by up-regulation of intramuscular fibrogenesis (fibrosis) and adipogenesis (fat infiltration)