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In a cell-based model induced by excessive dopamine, L-theanine exhibited neuroprotective effects on neuronal injury through the release of body fluid molecules from astrocytes, such as glutathione (36)

Among other genes involved in GSH metabolism, glutathione transferase M2-2 ( Gstm2 ), which is suggested to prevent the neurotoxic effects of DA oxidation on dopaminergic neurodegeneration in Parkinsons disease 31 , was significantly downregulated by 8 months (Fig

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