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These symptoms may affect nutrient absorption and electrolyte balance, and could also indicate serious complications such as pancreatitis or gallbladder disease

Although FoxO1 activates both MAFbx/Atrogin-1 and MuRF1 expression in cultured myotubes, the ability of FoxO1 to induce MuRF1 expression is independent of its DNA binding [33]

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Impaired mitochondrial function due to the downregulation of nuclear-encoded genes involved in oxidative phosphorylation increases ROS production, which in turn phosphorylates the serine residues of insulin receptors and IRSs, thereby resulting in SLCI and a reduction in IR

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