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"Structure of Trypanosoma brucei glutathione synthetase: domain and loop alterations in the catalytic cycle of a highly conserved enzyme"

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moreover, the exerted agents are influenced by a multiple of molecular and cellular pathways such as Mitogen-activated protein kinases (MAPK), Phosphatase and Tensin Homolog deleted on chromosome 10 (PTEN), signal transducer and activator of transcription (STAT), and a network of miRNA (Figure 6)

The increase in proinflammatory cytokines is hypothesized to deplete GSH to facilitate replication of SARS-CoV-2 and other viruses, leading to exacerbated symptoms [36]

In contrast to -glutamylcysteine synthetase, glutathione synthetase accepts a large variety of glutamyl-modified analogs of -glutamylcysteine, but is much more specific for cysteine-modified analogs of -glutamylcysteine