1 Introduction A growing number of cancer patients can survive due to chemotherapy, although it is unfortunate that those who undergo treatment often face adverse effects ( Although the pathophysiology of cisplatin-induced cognitive impairment (CICI) has not been fully elucidated, several pathophysiologic mechanisms, including oxidative stress, inflammation, mitochondrial dysfunction, DNA damage, and apoptosis, have been proposed as possible explanations ( Many studies have shown that cisplatin-induced mitochondrial damage in cells leads to oxidative stress and neuronal damage that results in cognitive impairment (CICI) ( 2 O 2 ) ( The GLP-1 and GLP-1 receptor pathway have been shown in previous studies to improve cognitive abilities significantly and to have neuroprotective effects by readily crossing the blood-brain barrier (BBB), minimizing oxidative stress, and reducing neuronal cell death in diabetes ( Despite the frequent occurrence of chemotherapy-induced cognitive impairment (chemobrain) in breast cancer patients and the widespread clinical use of cisplatin, there remains a significant gap in research addressing potential neuroprotective interventions, particularly the role of tirzepatide in attenuating cisplatin-induced memory deficits and oxidative stress
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