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The released SIM inhibits HMGCR, reducing downstream mevalonate pathway metabolites, such as IPP, resulting in GPX4 dysfunction, impaired lipid repair capacity, and LPO accumulation, ultimately inducing ferroptosis and establishing a comprehensive lipid metabolism regulatory axis

[DOI] [PubMed] [Google Scholar] 19.Teprotumumab for thyroid-associated ophthalmopathy

No vasculitis is found

Understanding this origin clarifies why GLP-1 therapy works so effectively for metabolic control