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These are significantly correlated with increased levels of inflammatory mediators such as C-reactive protein, increased aldehyde formation due to oxidative damage, increased nitric oxide production and hyper nityrosylation and low antioxidant levels (zinc, selenium, glutathione SH groups, glutathione peroxidase, total antioxidant capacity) (36)

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Antioxidants (Basel) 10 (3), 364

IL-1R1 and TLR4 are expressed in astrocytes and BBB ECs and have been shown to be upregulated in clinical and experimental studies of epilepsy