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This review will focus on pre-clinical data showing disturbed glutamatergic neurotransmission in cells and animal models of IEMs that may offer perspectives for the development of novel adjuvant treatments for these diseases
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Therefore, we concluded that DDIT4/PGC-1 is important for AMSCs regulating mitochondrial function in APAP-induced hepatotoxicity and that DDIT4/PGC-1 may serve as a molecular target for the treatment of AILI
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