Gastroenterology 84 , 840842 (1983)
It has been demonstrated that numerous cancers generate high levels of reactive oxygen species (ROS) compared to healthy tissue counterparts, where ROS levels are normally maintained in a tightly controlled manner.3 In T-ALL, ROS levels have been shown to be heightened, and this can inactivate phosphatase and the tensin homolog (PTEN), promoting leukemia cell survival.4 In human T-ALL, ROS levels are restrained by downregulation of protein kinase c theta (PKC) caused by NOTCH-1, a commonly activated mutation in T-ALL.5 However, if ROS stress levels are pushed above a certain threshold, cell death is forced to occur.3 Therefore, ROS promoting drugs may be an effective way of targeting cancer cells
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While regulatory bodies and pharmaceutical companies are discussing potential solutions to the drug shortages, societies such as the American Diabetes Association are providing guidelines to assist with alternative treatment options for patients with type 2 diabetes mellitus as well as guidelines to assist with dosage changes to be made while GLP-1 therapies are low in stock
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