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The interplay among multiple pathobiological pathways, such as the persistence of viral reservoirs, direct SARS-CoV-2-related tissue damage, dysbiosis, ongoing inflammation, autoimmunity and microthrombi phenomena, has been proposed as the main driver of Long COVID [9, 10]

[Experiences with L-carnitine in the post-stress phase]

Immunocytochemical studies suggest two pathways for enteroendocrine cell differentiation in the colon

But that's where the similarities end

The difference in distribution of carnitine and its derivatives between PBMC and erylysate in the present study with higher carnitine proportions in PBMC might be a consequence of different transport mechanisms as carnitine requirement of erythrocytes, cells without mitochondria, could be lower than in PBMC