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[55] It achieves glycemic control primarily through a glucose-dependent mechanism: by binding to GLP-1 receptors on beta cells, it elevates intracellular levels of cyclic AMP (cAMP) and activates protein kinase A (PKA) and related signaling pathways

doi: 10.1016/j.immuni.2021.04.022 98 WongCKMcLeanBABaggioLLKoehlerJAHammoudRRittigNet al

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By inhibiting beta-amyloid plaque formation, reducing neuronal oxidative damage, and dampening neuroinflammation (17, 18), it offers a multifaceted approach to brain health

Duration of Cycle: 30 days