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In a male mouse model of atherosclerosis, the versatile platform integrates cell-mediated natural targeting with adaptable size transformation for enhanced intraplaque penetration and unfavorable macrophage signaling reprogramming, offering opportunities for precise and multifaceted atherosclerosis therapy

6A), indicating the overlapping roles of these proteins in synaptic vesicle release and clathrin-mediated endocytosis

Differential S-nitrosylation of proteins in Alzheimers disease

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Fetal stress and programming of hypoxic/ischemic-sensitive phenotype in the neonatal brain: mechanisms and possible interventions